PMID:18225562

Citation	Sedliarou, I, Matsuse, M, Saenko, V, Rogounovitch, T, Nakazawa, Y, Mitsutake, N, Namba, H, Nagayama, Y and Yamashita, S Overexpression of wild-type THRbeta1 suppresses the growth and invasiveness of human papillary thyroid cancer cells. Anticancer Res. 27:3999-4009
Abstract	Thyroid hormone receptors (THRs) are transcription factors which regulate cell growth and differentiation. The compromised function of THRs has been reported in several human malignancies, suggesting their implication in carcinogenesis.
Links	PubMed
Keywords	Adenoviridae/genetics; Carcinoma, Papillary/enzymology; Carcinoma, Papillary/genetics; Carcinoma, Papillary/metabolism; Carcinoma, Papillary/pathology; Cell Cycle/physiology; Cell Growth Processes/physiology; Cell Line, Tumor; Humans; MAP Kinase Signaling System; Matrix Metalloproteinase 1/metabolism; Matrix Metalloproteinase 9/metabolism; Mitogen-Activated Protein Kinase 1/metabolism; Mitogen-Activated Protein Kinase 3/metabolism; Neoplasm Invasiveness; Proto-Oncogene Proteins c-akt/metabolism; Thyroid Hormone Receptors beta/biosynthesis; Thyroid Hormone Receptors beta/genetics; Thyroid Neoplasms/enzymology; Thyroid Neoplasms/genetics; Thyroid Neoplasms/metabolism; Thyroid Neoplasms/pathology; Transcriptional Activation; Transfection
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