PMID:10838136: Difference between revisions

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<span style = 'font-size: large'> '''Routledge, MN'''  (2000) Mutations induced by reactive nitrogen oxide species in the supF forward mutation assay.''Mutat. Res.'' '''450''':95-105</span>
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!align=left  |Citation
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'''No author listed'''  (2000) Mutations induced by reactive nitrogen oxide species in the supF forward mutation assay. ''Mutat. Res.'' '''450''':95-105
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!align=left  |Abstract
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Nitric oxide is an important bioregulatory molecule with a range of physiological functions. Nitric oxide can also react with oxygen species to produce a range of reactive nitrogen oxides that can damage DNA and lead to mutations of the DNA base sequence. The mutagenicity of a variety of reactive nitrogen oxide species and related DNA damaging agents in the supF assay are reviewed here, in the context of recent reports that relate to the nature of the DNA lesions responsible for the induced mutations. Mutations induced by nitric oxide in the supF assay are compared to those induced by N(2)O(3), nitrous acid, peroxynitrite and different reactive oxygen species. The effect of replication of the damaged pSP189 plasmid in human cells or Escherichia coli cells is also considered.
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!align=left  |Links
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[http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=10838136 PubMed]


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|{{TableHeadingColor}} |'''Abstract''' || Nitric oxide is an important bioregulatory molecule with a range of physiological functions. Nitric oxide can also react with oxygen species to produce a range of reactive nitrogen oxides that can damage DNA and lead to mutations of the DNA base sequence. The mutagenicity of a variety of reactive nitrogen oxide species and related DNA damaging agents in the supF assay are reviewed here, in the context of recent reports that relate to the nature of the DNA lesions responsible for the induced mutations. Mutations induced by nitric oxide in the supF assay are compared to those induced by N(2)O(3), nitrous acid, peroxynitrite and different reactive oxygen species. The effect of replication of the damaged pSP189 plasmid in human cells or Escherichia coli cells is also considered.
!align=left  |Keywords
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DNA Damage; DNA, Bacterial/drug effects; DNA, Bacterial/genetics; Escherichia coli/drug effects; Escherichia coli/genetics; Genes, Suppressor/drug effects; Humans; Mutation; Nitrates/toxicity; Nitric Oxide/toxicity; RNA, Transfer/genetics
|{{TableHeadingColor}} nowrap |'''Links''' ||  [http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=10838136 PubMed]
 
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==Significance {{HelpIcon|Help:Literature}} ==
==Significance {{HelpIcon|Help:Literature}} ==
{{LitSignificance}}
{{LitSignificance}}

Revision as of 17:53, 1 September 2013

Citation

No author listed (2000) Mutations induced by reactive nitrogen oxide species in the supF forward mutation assay. Mutat. Res. 450:95-105

Abstract

Nitric oxide is an important bioregulatory molecule with a range of physiological functions. Nitric oxide can also react with oxygen species to produce a range of reactive nitrogen oxides that can damage DNA and lead to mutations of the DNA base sequence. The mutagenicity of a variety of reactive nitrogen oxide species and related DNA damaging agents in the supF assay are reviewed here, in the context of recent reports that relate to the nature of the DNA lesions responsible for the induced mutations. Mutations induced by nitric oxide in the supF assay are compared to those induced by N(2)O(3), nitrous acid, peroxynitrite and different reactive oxygen species. The effect of replication of the damaged pSP189 plasmid in human cells or Escherichia coli cells is also considered.

Links

PubMed

Keywords

DNA Damage; DNA, Bacterial/drug effects; DNA, Bacterial/genetics; Escherichia coli/drug effects; Escherichia coli/genetics; Genes, Suppressor/drug effects; Humans; Mutation; Nitrates/toxicity; Nitric Oxide/toxicity; RNA, Transfer/genetics

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