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acnB:Gene
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Allele | Nt change(s) | AA change(s) | Phenotype: Type | Phenotype: Description | Reference | Availability | Comments | |
---|---|---|---|---|---|---|---|---|
public |
acnB(del) (Keio:JW0114) |
deletion |
deletion |
PMID:16738554 |
||||
public |
acnB(del)::kan |
deletion |
Biolog:respiration |
unable to respire Bromosuccinate |
PMID:16095938 |
|||
public |
acnB(del)::kan |
deletion |
Biolog:respiration |
unable to respire L-Asparagine |
PMID:16095938 |
|||
public |
acnB(del)::kan |
deletion |
Biolog:respiration |
unable to respire L-Aspartate |
PMID:16095938 |
|||
public |
acnB(del)::tetR |
Internal deletion of acnB replaced with tetR gene. |
Growth Phenotype |
Cannot grow on acetate as the sole carbon and energy source with or without supplementation with glutamate. |
PMID:9202458 |
|||
public |
acnB(del)::tetR |
Internal deletion of acnB substituted with tetR. |
Growth Phenotype |
Grows poorly on minimal glucose medium unless the medium is supplemented with glutamate. The same phenotype is seen for growth on minimal medium with glycerol, lactate, pyruvate, or succinate as the carbon and energy source. |
PMID:9202458 |
Mutants lacking citrate synthase (gltA) or isocitrate dehydrogenase (icd) are glutamate auxotrophs. The mutant lacking acnB doesn't have an absolutely requirement for glutamate because of the second aconitase activity (acnA). | ||
public |
acnB736(del)::kan |
PMID:16738554 |
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